{"id":20800,"date":"2019-01-23T15:59:13","date_gmt":"2019-01-23T23:59:13","guid":{"rendered":"https:\/\/sop.washington.edu\/?p=20800"},"modified":"2019-01-23T15:59:13","modified_gmt":"2019-01-23T23:59:13","slug":"undaunted-by-setbacks-jash-unadkat-and-his-lab-team-search-for-new-therapies-to-fight-alzheimers","status":"publish","type":"post","link":"https:\/\/staff.washington.edu\/twentzel\/wordpress\/undaunted-by-setbacks-jash-unadkat-and-his-lab-team-search-for-new-therapies-to-fight-alzheimers\/","title":{"rendered":"Undaunted by setbacks, Jash Unadkat and his lab team search for new therapies to fight Alzheimer\u2019s"},"content":{"rendered":"
\"Tot
Tot Nguyen and Jash Unadkat collaborate to find new therapies against Alzheimer\u2019s\u2014now with quantitative proteomics (not shown here). <\/figcaption><\/figure>\n

It\u2019s called an experiment for a reason. The secret in science\u2014what world-class researchers know\u2014is that you don\u2019t let the failures stop you. You think of the failures as more data, part of your larger body of work.<\/p>\n

For scientists as creative and productive as Jash Unadkat<\/b>, learning to move past the failures, to learn from them and continue to what\u2019s next, is a key skill that he encourages in his graduate students, post docs, and staff. \u201cWe have to pick ourselves up, dust ourselves off, and march on\u2014like a child learning to walk,\u201d he reminds them.<\/p>\n

A few years ago he and his lab team thought they had a big break through in a novel approach to treating Alzheimer\u2019s disease. In the brain, there is a key transporter\u00a0protein (P-glycoprotein) that keeps drugs from getting through the blood brain barrier. \u201cIt acts like a bouncer at a nightclub,\u201d said Jash. \u201cIt keeps some drugs from getting in, but it also ejects \u03b2-amyloid from the brain.\u201d He continued, \u201cWe hypothesized a number of years ago that maybe the reason you get Alzheimer\u2019s is due to an accumulation of \u03b2-amyloid in brain and they clump and form plaques.\u201d<\/p>\n

They thought further that perhaps one of the reasons \u03b2-amyloid builds up in the brain is because the \u201cbouncer\u201d P-glycoprotein is compromised, and not able to kick enough of the \u03b2-amyloid out of the so-called Brain Club. The team decided to introduce a drug, Rifampin, that increases the amount of P-glycoprotein in the intestine to see if it would do the same in the brain and improve the protein\u2019s ability to remove \u03b2-amyloid. To their disappointment they found that Rifampin did not increase the activity level of P-glycoprotein in the brain.<\/p>\n

\n

\u201cWe were so optimistic that we had a solution that could lead to using an already approved drug in a new way to treat Alzheimer\u2019s disease. Our findings were disappointing, but our work continues with new methods.\u201d \u2014Jash Unadkat, Milo Gibaldi Endowed Professor of Pharmaceutics<\/span><\/p>\n<\/blockquote>\n

Although the team published the research and (then) graduate student Li Liu <\/b>received a Best Paper Award in DMD, they were now back to square one in the riddle. They knew that drug didn\u2019t work and didn\u2019t know if the lower level of P-glycoprotein was a cause or effect of Alzheimer\u2019s disease. Well, or maybe square two.<\/p>\n

Despite that drug not working, it did advance the research and led to an award-winning paper for Anand Deo<\/b>, one of Jash\u2019s post doc fellows at the time. A few years have passed and the team is taking a new approach with quantitative\u00a0proteomics o investigate the effect of Alzheimer\u2019s disease on P-glycoprotein levels in the human brain by using liquid chromatography and mass spectrometry and to see if they can measure the difference in P-glycoprotein using this other approach.<\/p>\n

They are looking to other proteins that may be important in the process. Proteomics will help by giving insight into the mechanisms in the cells that can change the P-glycoprotein activity levels. There is comfort in the negative results of their initial study. \u201cIt means that if someone is taking Rifampin they are not going to increase P-glycoprotein activity in brain, which is particularly important if they are taking a drug for their central nervous system disease, like depression or epilepsy. They know they can safely take the drug because of this study,\u201d said Jash.\u00a0<\/span><\/p>\n

 <\/p>\n

To study faculty like Dr. Jash Unadkat<\/strong>, click on the link for\u00a0more information about our\u00a0PhD Program in Pharmaceutics<\/a>.<\/p>\n

Link to Pharmaceutics archived news<\/a><\/p>\n","protected":false},"excerpt":{"rendered":"

It\u2019s called an experiment for a reason. The secret in science\u2014what world-class researchers know\u2014is that you don\u2019t let the failures stop you. You think of the failures as more data, part of your larger body…<\/p>\n","protected":false},"author":3,"featured_media":20801,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"_monsterinsights_skip_tracking":false,"_monsterinsights_sitenote_active":false,"_monsterinsights_sitenote_note":"","_monsterinsights_sitenote_category":0,"footnotes":""},"categories":[33],"tags":[],"class_list":["post-20800","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-pceut-news"],"yoast_head":"\nUndaunted by setbacks, Jash Unadkat and his lab team search for new therapies to fight Alzheimer\u2019s - School of Pharmacy<\/title>\n<meta name=\"description\" content=\"It\u2019s called an experiment for a reason. 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